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2017 Apr;55(4):260-266

Primary and immortalised adipocyte models document effects on lipolysis (enhanced by glucagon receptor activation), browning markers (uncoupling protein 1, PGC-1), and thermogenic gene expression

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The findings in mouse models of SOD1 ALS show an increase of complement activators (C1qB, C4, C3, C5, and CD88) and decrease in complement regulators (CD55, CD59a) during disease progression in spinal-cord-tissue supported investigations of complement inhibitors in ALS [256]